September 2024
Volume 24, Issue 10
Open Access
Vision Sciences Society Annual Meeting Abstract  |   September 2024
Reduced visual acuity due to defocus cannot fully account for the abnormal fixational eye movements of persons with amblyopia
Author Affiliations & Notes
  • Sunwoo Kwon
    Herbert Wertheim School of Optometry and Vision Science, UC Berkeley
    Center for Innovation in Vision and Optics, UC Berkeley
    Exponent
  • Julien Belen
    Herbert Wertheim School of Optometry and Vision Science, UC Berkeley
  • Jessica Lien
    Herbert Wertheim School of Optometry and Vision Science, UC Berkeley
  • Artashes Yeritsyan
    Herbert Wertheim School of Optometry and Vision Science, UC Berkeley
  • Nam Do
    Herbert Wertheim School of Optometry and Vision Science, UC Berkeley
  • Dennis Levi
    Herbert Wertheim School of Optometry and Vision Science, UC Berkeley
    Center for Innovation in Vision and Optics, UC Berkeley
    Helen Wills Neuroscience Institute, UC Berkeley
  • Footnotes
    Acknowledgements  Center for Innovation in Vision and Optics (CIVO) Fellowship, NEI R21
Journal of Vision September 2024, Vol.24, 708. doi:https://doi.org/10.1167/jov.24.10.708
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      Sunwoo Kwon, Julien Belen, Jessica Lien, Artashes Yeritsyan, Nam Do, Dennis Levi; Reduced visual acuity due to defocus cannot fully account for the abnormal fixational eye movements of persons with amblyopia. Journal of Vision 2024;24(10):708. https://doi.org/10.1167/jov.24.10.708.

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      © ARVO (1962-2015); The Authors (2016-present)

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Abstract

Amblyopia is associated with impaired visual acuity, abnormal processes in primary visual cortex (V1), and reduced coordination of activity in binocular neurons. Recent studies have demonstrated that these abnormalities extend beyond V1 with potential impacts on higher order processing and in oculomotor areas that affect visually-guided behaviors. While large fixational instabilities found in amblyopic eyes are highly correlated with worsening acuity in these patients, no such correlation was found in a neurotypical population (Raveendran et al., 2019). It is unclear whether the poor acuity causes fixational instability or vice versa or if the two are correlated as a byproduct of abnormal processes in V1. Therefore, we investigated whether visual acuity is truly a limiting factor in oculomotor and visually-guided behavior. We simulated reduced visual acuity observed in the amblyopic eye in the non-dominant eyes (NDEs) of corrected-to-normal neurotypical observers by applying convex lenses to induce retinal-defocus while the observers viewed naturalistic stimuli – a filtered version of “Where’s Waldo” – while performing a visual search task. We tested a set of logMAR acuity differences (0.2, 0.4, 0.6, and 0.8) as observed in amblyopic patients and compared the oculomotor behavior of the normal eye with lens-induced acuity loss in the following conditions: monocular NDE and binocular viewing. We observed lens induced acuity loss had a significant impact on the observer’s drift magnitude and fixational instabilities compared to the no-blur condition in the NDE; however, further reductions in visual acuity did not have any significant impact. Binocular viewing significantly improved fixational instability and reduced reaction time when compared with monocular viewing in both amblyopes and neurotypicals; this effect persisted as a function of retinal-defocus induced reduction in visual acuity. Our results demonstrate that the reduced visual acuity of patients with amblyopia cannot be fully accounted for by abnormal fixational eye movements.

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